Tissue-level inflammation and ventricular remodeling in hypertrophic cardiomyopathy
نویسندگان
چکیده
منابع مشابه
Progressive left ventricular remodeling in patients with hypertrophic cardiomyopathy and severe left ventricular hypertrophy.
OBJECTIVES The aim of this study was to determine the natural history of patients with hypertrophic cardiomyopathy (HCM) and severe left ventricular hypertrophy (LVH) (i.e., maximal left ventricular wall thickness [MLVWT] >/=30 mm) and whether changes in cardiac morphology influence the course of the disease. BACKGROUND Severe LVH is common in young and rare among elderly patients with HCM. T...
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Heart failure in the setting of reduced systolic pump function, if left untreated, is characterized by progressive ventricular dilation and dysfunction. Many of the major advances in pharmacologic treatments for heart failure achieved over the last two decades came with the recognition that this process, referred to as ventricular remodeling, results from abnormal mechanical stress on the myoca...
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BACKGROUND Alcohol septal ablation (ASA) reduces left ventricular outflow tract (LVOT) pressure gradient in patients with hypertrophic obstructive cardiomyopathy (HOCM), which leads to left ventricular remodeling. We sought to describe the early to midterm changes and modulating factors of the remodeling process using cardiac MRI (CMR). METHODS AND RESULTS CMR was performed at baseline and 1 ...
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BACKGROUND In hypertrophic cardiomyopathy (HCM), the degree of left ventricular hypertrophy (LVH) could influence the development of ventricular arrhythmias. OBJECTIVE In HCM, analyze the association between the occurrence of ventricular arrhythmias determined by Holter electrocardiogram (ECG-Holter) and the degree of LVH determined by maximum wall thickness (MWT) in echocardiography and body...
متن کاملLeft ventricular hypertrophy and hypertrophic cardiomyopathy.
eft ventricular myocardial hypertrophy (LVH) occurs in any conditions, usually, it is thought, as a secondary or compensatory” response to abnormal pressure or volume oading and to stimuli, as yet incompletely understood, esulting from deficiencies in cardiac performance (1). hese responses may be modulated by genetic predisposiions, a relationship also not well understood. “Compensaory” hypert...
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ژورنال
عنوان ژورنال: Journal of Thrombosis and Thrombolysis
سال: 2020
ISSN: 0929-5305,1573-742X
DOI: 10.1007/s11239-019-02026-1